Vaccination with Receptor Protein defend Mice against AD

A soluble version of a protein inculpated in the formation of toxic amyloid plaques was shown to prevent assemblage of amyloid beta (Abeta) and slow evolution of Alzheimer’s disease (AD) in a mouse model. The brains of Alzheimer’s disease patients have been shown to express raised levels of Toll-like receptors (TLRs), a family of proteins on the surface of immune cells that recognize molecules released by pathogens or damaged cells and then initiate an appropriate immune response.
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